Duchenne Muscular Dystrophy Quiz

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Test your DMD knowledge.

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samah Almehmadi

Arabia Saudí

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Duchenne Muscular Dystrophy Quiz


Froggy Jumps Duchenne Muscular Dystrophy QuizVersión en línea Test your DMD knowledge. por samah Almehmadi 1 What protein is lacking in Duchenne muscular dystrophy? a Dystrophin b Actin c Myoglobin 2 DMD predominantly affects which gender due to its inheritance? a Males b Females c Non-binary 3 What happens to the sarcolemma when dystrophin is absent? a Thickens without damage b Tears during muscle contraction c Becomes stronger 4 What is a key consequence of Ca++ influx in DMD muscle fibers? a Cellular damage and fiber degradation b Increased endurance c Enhanced muscle growth 5 Which body system is progressively affected in later stages of DMD? a Respiratory and circulatory muscles b Digestive system c Skeletal bones 6 DMD is caused by a mutation in the gene coding for which protein? a Dystrophin b Collagen c Utrophin 7 What is the typical prognosis for untreated DMD? a Death due to respiratory failure in the 20s b Recovery in adolescence c Stable condition for life 8 Why was myoblast therapy largely unsuccessful in DMD? a They cured other diseases instead b They caused immune rejection only c Difficulty delivering the gene and cells to muscles 9 What recent strategy aims to compensate for dystrophin loss? a Reducing calcium intake b Boosting utrophin production c Replacing muscles with steel 10 DMD usually first presents with which symptom? a Difficulty with balance and motion b Vision loss c Seizures 11 Duchenne muscular dystrophy (DMD) is caused by a lack of which protein? a Actin b Dystrophin c Tubulin 12 Which chromosome is typically involved in DMD inheritance? a Chromosome 21 b X chromosome c Y chromosome 13 DMD primarily affects which gender? a Non-binary individuals b Females c Males 14 What happens to the sarcolemma due to dystrophin deficiency? a It becomes impermeable to Ca++ b It thickens significantly c It tears during contractions 15 Ca++ influx in muscle fibers leads to: a Healing and growth of fibers b Cellular damage and fiber degradation c Increased muscle strength 16 What is the usual prognosis for life expectancy in DMD without advanced care? a Lifespan unaffected b Live into 60s c Most do not live past their 20s 17 Which bodily systems are affected as DMD progresses beyond the legs? a Digestive system only b Nervous system exclusively c Breathing and circulation muscles 18 What protein is being explored to compensate for dystrophin loss? a Utrophin b Collagen c Myosin 19 Why were myoblast therapies largely unsuccessful in humans? a They cause excessive dystrophin production b Difficulty delivering functional dystrophin-producing cells c They cure the disease instantly 20 What is a key reason utrophin-based strategies are pursued? a Utrophin blocks muscle growth b Utrophin may substitute for dystrophin and prevent damage c Utrophin causes more damage

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Duchenne Muscular Dystrophy Quiz

Froggy Jumps

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Froggy Jumps

Duchenne Muscular Dystrophy QuizVersión en línea

por samah Almehmadi

What protein is lacking in Duchenne muscular dystrophy?

DMD predominantly affects which gender due to its inheritance?

What happens to the sarcolemma when dystrophin is absent?

What is a key consequence of Ca++ influx in DMD muscle fibers?

Which body system is progressively affected in later stages of DMD?

DMD is caused by a mutation in the gene coding for which protein?

What is the typical prognosis for untreated DMD?

Why was myoblast therapy largely unsuccessful in DMD?

What recent strategy aims to compensate for dystrophin loss?

DMD usually first presents with which symptom?

Duchenne muscular dystrophy (DMD) is caused by a lack of which protein?

Which chromosome is typically involved in DMD inheritance?

DMD primarily affects which gender?

What happens to the sarcolemma due to dystrophin deficiency?

Ca++ influx in muscle fibers leads to:

What is the usual prognosis for life expectancy in DMD without advanced care?

Which bodily systems are affected as DMD progresses beyond the legs?

What protein is being explored to compensate for dystrophin loss?

Why were myoblast therapies largely unsuccessful in humans?

What is a key reason utrophin-based strategies are pursued?