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NCLEX MI & Vascular Quiz

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MI and peripheral vascular disorders essentials.

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NCLEX MI & Vascular Quiz
 

NCLEX MI & Vascular QuizVersión en línea

MI and peripheral vascular disorders essentials.

por Danielle Kendricks
1

What is the primary consequence of a completely occluded coronary artery?

2

What decreases after MI due to reduced contractility?

3

Classic MI pain is best described as?

4

Which symptom is more common in women with MI?

5

Troponin is considered what in MI?

6

STEMI on ECG shows what finding?

7

NSTEMI ECG usually shows?

8

Initial medical management for acute MI is often abbreviated as MONA. Which is correct order variant?

9

Which drug class is used in AMI to reduce workload and remodeling?

10

What is a key reperfusion goal for PCI after MI?

11

Thrombolytics are used when PCI is unavailable. This is to?

12

Which finding increases risk for cardiogenic shock after MI?

13

Nursing priority after MI includes monitoring for which complication?

14

S3 sound after MI may indicate?

15

What non-pharmacologic measure supports MI recovery?

16

Chronic Venous Disease primarily involves which process?

17

What symptom is typical of Chronic Venous Disease?

18

Which diagnostic checks vein function & reflux?

19

Foam sclerotherapy is a treatment for what?

20

What is a primary non-pharmacologic measure for CVD?

21

Which is a common procedure for varicose veins?

22

Nursing management for CVD includes?

Feedback

Occlusion causes irreversible necrosis if blood flow isn't restored.

Contractility loss lowers stroke volume and CO.

Classic MI pain persists despite rest and nitro.

Atypical MI in women includes fatigue and GI symptoms.

Troponin elevation is highly specific for myocardial injury.

ST elevation indicates STEMI requiring urgent reperfusion.

NSTEMI lacks ST elevation; may have depression or T inversion.

MONA themes can vary; commonly Morphine, Oxygen, Nitroglycerin, Aspirin in sequence.

ACE inhibitors help after MI to prevent remodeling.

PCI should be performed quickly to minimize myocardial damage.

Thrombolytics dissolve clots to reflow ischemic tissue when PCI cannot be done.

Cardiogenic shock involves hypotension and reduced perfusion.

Dysrhythmias are common and dangerous post-MI.

S3 suggests volume overload and possible HF.

Monitoring detects arrhythmias early; bed rest is initially advised.

VALVE incompetence causes reflux and venous stasis.

Venous edema tends to accumulate with prolonged standing or day progression.

Duplex ultrasound assesses valve function and reflux in veins.

Sclerotherapy targets varicose veins to close abnormal veins.

Leg elevation reduces venous pressure and edema.

Laser ablation closes dilated veins with minimal invasion.

Compression therapy and skin care reduce ulcers and edema.

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